Home / HIVEP1 Knockout Cell Lines

HIVEP1 Knockout Cell Lines

Gene: HIVEP1

Official Full Name: HIVEP zinc finger 1provided by HGNC

Gene Summary: This gene encodes a transcription factor belonging to the ZAS family, members of which are large proteins that contain a ZAS domain - a modular protein structure consisting of a pair of C2H2 zinc fingers with an acidic-rich region and a serine/threonine-rich sequence. These proteins bind specifically to the DNA sequence motif, GGGACTTTCC, found in the enhancer elements of several viral promoters, including human immunodeficiency virus (HIV), and to related sequences found in the enhancer elements of a number of cellular promoters. This protein binds to this sequence motif, suggesting a role in the transcriptional regulation of both viral and cellular genes. [provided by RefSeq, Oct 2011]

Get A Quote
Products Background

Products

Catalog Number Product Name Species Gene Passage ratio Mycoplasma testing Price
KO37380 HIVEP1 Knockout cell line (HeLa) Human HIVEP1 1:3~1:6 Negative Online Inquiry
KO37381 HIVEP1 Knockout cell line (HCT 116) Human HIVEP1 1:2~1:4 Negative Online Inquiry
KO37382 HIVEP1 Knockout cell line (HEK293) Human HIVEP1 1:3~1:6 Negative Online Inquiry
KO37383 HIVEP1 Knockout cell line (A549) Human HIVEP1 1:3~1:4 Negative Online Inquiry

Background

HIVEP1 Gene Knockout Cell Lines are engineered cell lines in which the HIVEP1 gene has been selectively disrupted through targeted gene editing techniques, such as CRISPR-Cas9. HIVEP1 (Human Immunodeficiency Virus Type 1 Enhancer Binding Protein 1) is a crucial transcription factor implicated in various cellular processes, including gene expression regulation, immune response modulation, and oncogenesis. The knockout of this gene provides researchers with a powerful tool to elucidate its role in biological pathways and to explore its involvement in disease mechanisms.

The primary function of HIVEP1 Gene Knockout Cell Lines lies in their ability to serve as model systems for studying the effects of HIVEP1 loss on cellular behavior, signaling pathways, and phenotypic outcomes. By utilizing these cell lines, scientists can investigate the gene's role in cancer progression, immune responses, and viral infections, offering insights that may lead to the development of novel therapeutic approaches. Moreover, knockout cell lines are invaluable resources for preclinical studies, enabling high-throughput screening of drug candidates and the identification of potential biomarkers.

In a landscape filled with various genetic tools, HIVEP1 Gene Knockout Cell Lines stand out due to their specificity and reliability. Unlike temporary knockdown approaches that yield transient effects, these stable knockout cell lines provide consistent and reproducible results, allowing for long-term studies. Researchers benefit from the enhanced ability to investigate gene functions in dynamic biological contexts without the confounding effects of transient expression variability.

The value of HIVEP1 Gene Knockout Cell Lines to researchers and clinicians cannot be overstated, as they not only facilitate deeper understanding of essential biological mechanisms but also drive innovation in therapeutic development. Furthermore, this product aligns with the growing thrust towards precision medicine by enabling tailored research that could inform personalized treatment strategies.

Our expertise at [Your Company] in developing cutting-edge biological products ensures that HIVEP1 Gene Knockout Cell Lines are produced with the utmost precision and quality. We are committed to supporting researchers and clinicians in their quest for scientific breakthroughs, providing them with the tools necessary for advancing biomedical research and improving patient outcomes.

Please note that all services are for research use only. Not intended for any clinical use.

Get a free quote

If your question is not addressed through these resources, you can fill out the online form below and we will answer your question as soon as possible.

0

There is no product in your cart.